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[DOI] [PubMed] [Google Scholar] 68.Hochwald SN, Rose DM, Brennan MF, Burt ME
Thus, the findings reveal a potential therapeutic avenue for RIPK1-related diseases through manipulating HK2 non-metabolic function
This suggests that components involved in TJ disassembly may represent useful therapeutic targets by limiting the impact of enteric pathogens on intestinal barrier integrity and restricting their systemic spread
JX06, a synthetic inhibitor, induces cytotoxicity by covalently modifying a hydrophobic pocket adjacent to the ATP binding site of PDK [188]
EBioMedicine 43 , 632640 (2019)
