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doi:10.1111/j.1432-1033.1982.tb05780.x

doi: 10.3390/cancers11070990 490 YangSWangCLiXWuCLiuCXueZet al

There are racial differences as well

10.1111/febs.15225 FEBS J

Broader solid tumor studies corroborate that pharmacological inhibition of CAF-mediated Gln synthesis disrupts tumor metabolic fitness, suggesting combinatorial targeting of tumoral and stromal glutamine metabolism, particularly through dual GLS1/ECM pathway inhibition, may offer promising therapeutic avenues for advanced prostate cancer (196)